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Hokkaido University Collection of Scholarly and Academic Papers (53.866 recursos)
HUSCAP (Hokkaido University Collection of Scholarly and Academic Papers) contains peer-reviewed journal articles, proceedings, educational resources and any kind of scholarly works of Hokkaido University.

Mostrando recursos 1 - 18 de 18

1. Caveolin-3 gene mutation in Japanese with rippling muscle disease - Yabe, Ichiro; Kawashima, A; Kikuchi, S; Higashi, T; Fukazawa, T; Hamada, T; Sasaki, H; Tashiro, K; ??, ??
Objectives: Rippling muscle disease (RMD) is a rare myopathy characterized by percussion-induced rapid muscle contractions, muscle mounding, and rippling.

2. Four mutations of the spastin gene in Japanese families with spastic paraplegia - Basri, Rehana; Yabe, Ichiro; Soma, Hiroyuki; Takei, Asako; Nishimura, Hiroyuki; Machino, Yuka; Kokubo, Yasumasa; Kosugi, Masafumi; Okada, Ryuichirou; Yukitake, Motohiro; Tachibana, Hisao; Kuroda, Yasuo; Kuzuhara, Shigeki; Sasaki, Hidenao
The most common form of HSP is caused by mutations of the spastin gene (SPAST), located on chromosome 2p21-p22, which encodes spastin, one of the ATPases associated with diverse cellular activities (AAA).

3. Focal fractionated radiotherapy for intramedullary spinal arteriovenous malformations: 10-year experience. - Hida, Kazutoshi; Shirato, Hiroki; Isu, Toyohiko; Seki, Toshitaka; Onimaru, Rikiya; Aoyama, Hidefumi; Ushikoshi, Satoshi; Miyasaka, Kazuo; Iwasaki, Yoshinobu
Candidates for this study were patients who experienced symptoms due to an intramedullary AVM but wereineligible for embolization or surgery.

4. Developmental expression of GABA transporter-1 and 3 during formation of the GABAergic synapses in the mouse cerebellar cortex. - Takayama, Chitoshi; Inoue, Yoshiro
In the brain, ?-amino butyric acid (GABA), released extrasynaptically and synaptically from GABAergic neurons, plays important roles in morphogenesis, expression of higher functions and so on.

5. Heredity in Multiple System Atrophy - Soma, Hiroyuki; Yabe, Ichiro; Takei, Asako; Fujiki, Naoto; Yanagihara, Tetsuro; Sasaki, Hidenao; ??, ??
We investigated the family histories of 157 Japanese patients with probable orpossible multiple system atrophy (MSA).

6. Nonsuture dural repair using polyglycolic acid mesh and fibrin glue: clinical application to spinal surgery - Hida, Kazutoshi; Yamaguchi, Satoshi; Seki, Toshitaka; Yano, Shunsuke; Akino, Minoru; Terasaka, Shunsuke; Uchida, Takanori; Iwasaki, Yoshinobu; ??, ??
We report our experience with a non-suture dural repair technique inpatients undergoing spinal surgery; it employs a dura substitute composed of polyglycolic acid(PGA) mesh and fibrin glue.

7. 5-HT1A receptor agonist affects fear conditioning through stimulations of the postsynaptic 5-HT1A receptors in the hippocampus and amygdala - Li, XiaoBai; Inoue, Takeshi; Abekawa, Tomohiro; Weng, ShiMin; Nakagawa, Shin; Izumi, Takeshi; Koyama, Tsukasa
Evidence from preclinical and clinical studies has shown that 5-HT1A receptor agonists have anxiolytic actions.

8. MSA-C is the predominant clinical phenotype of MSA in Japan : Analysis of 142 patients with probable MSA - Yabe, Ichiro; Soma, Hiroyuki; Takei, Asako; Fujiki, Naoto; Yanagihara, Tetsuro; Sasaki, Hidenao
We investigated the clinical features and mode of disease progression in 142 patients with probable multiple system atrophy (MSA) according to the Consensus Criteria.

9. Effect of carbamazepine on the single oral dose pharmacokinetics of perospirone and its active metabolite - Masui, Takuya; Kusumi, Ichiro; Takahashi, Yoshito; Koyama, Tsukasa
Perospirone is a serotonin 5-HT2A and dopamine D2 receptor antagonist which originated in Japan.

10. Developmental localization of potassium chloride co-transporter 2 in granule cells of the early postnatal mouse cerebellum with special reference to the synapse formation - Takayama, C.; Inoue, Y.
To clarify the time-course of changes in the GABA actions during development, we examined the developmental localization of the KCC2 in the granule cells of the postnatal mouse cerebellum using specific antibodies against KCC2.

11. Formation of GABAergic synapses in the cerebellum - Takayama, Chitoshi
In the cerebellum, formation of GABAergic synapses is crucial for cerebellar functions.

12. Extrasynaptic localization of GABA in the developing mouse cerebellum - Takayama, Chitoshi; Inoue, Yoshiro
In the adult brain, ?-amino butyric acid (GABA) is synaptically released and mediates inhibitory transmission.

13. Morphological development and maturation of the GABAergic synapses in the mouse cerebellar granular layer - Takayama, Chitoshi; Inoue, Yoshiro
In the adult central nervous system (CNS), ?-amino butyric acid (GABA) is a predominant inhibitory neurotransmitter, which regulates glutamatergic activity.

14. Transient expression of GABAA receptor ?2 and ?3 subunits in differentiating cerebellar neurons - Takayama, Chitoshi; Inoue, Yoshiro
To clarify how GABA exerts its effect on immature neurons, we examined the expression of the GABAA receptor ?2 and ?3 subunits, which are abundantly expressed before ?1 and ?6 subunits appear, in the developing mouse cerebellum using in situ hybridization.

15. Normal formation of the postsynaptic elements of GABAergic synapses in the reeler cerebellum - Takayama, Chitoshi; Inoue, Yoshiro
Synaptic transmission mediated by ?-amino butyric acid (GABA) plays an important role in inhibition of glutamatergic excitatory transmission and expression of higher brain functions, such as memory, learning and anxiety.

16. GABAergic signaling in the developing cerebellum - Takayama, Chitoshi; Inoue, Yoshiro
The definitive version is available at www.blackwell-synergy.com.

17. Developmental localization of potassium chloride co-transporter 2 (KCC2) in the Purkinje cells of embryonic mouse cerebellum - Takayama, Chitoshi; Inoue, Yoshiro
To clarify the time-course of the developmental shift on the Purkinje cells, we examined KCC2-localization in the embryonic mouse cerebellum.

18. GABAergic signaling in the developing cerebellum - Takayama, Chitoshi
Furthermore, normal formation ofGABAergic synapses is crucial for the expression of higher brain functions such as memory,learning and motor coordination, and various psychiatric diseases such as anxiety disorders,epilepsy, schizophrenia and autism are partly caused by the dysfunction of GABA in thedeveloping and mature brain.